By Ramana S. Moorthy, MD
A dialogue of the medical method of uveitis ends up in greatly rewritten chapters on noninfectious (autoimmune) and infectious types of uveitis. additionally coated are endophthalmitis, masquerade syndromes, and problems of uveitis. A dialogue on ocular involvement in AIDS has been up to date. The part on immunology describes the human immune reaction in phrases that make it hugely available to readers.
Read Online or Download 2008-2009 Basic and Clinical Science Course: Section 9: Intraocular Inflammation and Uveitis (Basic and Clinical Science Course 2008-2009) PDF
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Additional info for 2008-2009 Basic and Clinical Science Course: Section 9: Intraocular Inflammation and Uveitis (Basic and Clinical Science Course 2008-2009)
In addition, some LCs leave the skin, enter the draining node, and encounter memory T lymphocytes there. Processing during the secondary response is much quicker, and within 24 hours restimulated memory cells enter the circulation and migrate to the 29 30 . Intraocular Inflammation and Uveitis toxin-exposed cutaneous site. Because abundant toxin remains, additional T Iymphocyte-LC stimulation occurs, inducing vigorous T-Iymphocyte cytokine production. The inflammatory mediators, in turn, recruit neutrophils and monocytes, leading to a severe inflammatory reaction within 12-36 hours after exposure, causing the typical epidermal blisters of poison ivy.
Eosinophils and neutrophils appear to be absent. Under various clinical or experimental conditions, however, high densities of T lymphocytes, B lymphocytes, macrophages, and neutrophils can infiltrate the choroid, choriocapillaris, and retina. The RPE and various cell types within the retina and the choroid (eg, pericytes) can synthesize many different cytokines (eg, TGF-~) that may alter the subsequent immune response. Local immune processing does not appear to occur. See also BCSC Section 12, Retina and Vitreous.
Class I molecules are present on almost all nucleated cells, indicating that most cells have the potential to stimulate CDS T lymphocytes. The CDS T-lymphocyte antigen receptor must recognize its own class I type before it can respond to tumor or viral antigens on the appropriate target cell, and therefore CDS T lymphocytes from 1 individual will not respond to a target cell 1 2 HLA class I molecule Infected APC 4 3 Costimulation Antigen receptor CD8 T Lymphocyte Figure 2-3 Class I-dependent antigen-presenting cells (APCs).
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