By Ramana S. Moorthy MD

ISBN-10: 1615251162

ISBN-13: 9781615251162

Starts off with an in-depth review of immunemedicated eye disorder, summarizing easy immunologic options, ocular immune responses and distinctive themes in ocular immunology. Discusses the scientific method of uveitis and stories noninfectious (autoimmune) and infectious sorts of uveitis, with an increased part on viral uveitis and new fabric on infectious and noninfectious scleritis. superior detection of infectious brokers by way of immunologic and genetic equipment and new biologic therapeutics are distinct. additionally covers endophthalmitis, masquerade syndromes, problems of uveitis and ocular points of AIDS. features a variety of new colour photos. significant revision 2011-2012

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Extra info for 2011-2012 Basic and Clinical Science Course, Section 9: Intraocular Inflammation and Uveitis (Basic & Clinical Science Course)

Example text

These include hydrolytic enzymes, elastase, metalloproteinases, gelatinase, myeloperoxidase, vitamin B 12 - binding protein, cytochrome b558 , and others. Granule contents are considered to remain inert and membrane-bound when the granules are intact, but they become active and soluble when granules fuse to the phagocytic vesicles or plasma membrane. An example of the effect of neutrophil-derived granule products is collagenase; collagenases are thought to contribute to corneal injury and liquefaction during bacterial keratitis and scleritis, especially in Pseudomonas infections.

In general. class I APCs are best for processing peptide antigens that have been synthesized by the host cell itself. including most tumor peptides or viral peptides after host cell invasion. MHC class II molecules (ie. HLA-DR. -DP. and -DQ) serve as the antigen-presenting platform for CD4. or helper. T lymphocytes (Fig 2-3). All APCs for CD4 T lymphocytes must express the MHC class II molecule. and the antigen receptor on the helper T lymphocyte can recognize peptide antigens only if they are presented with class II molecules Simultaneously.

PAFs also serve physiologic functions unrelated to inflammation, especially in reproductive biology, the physiology of secretory epithelium, and neurobiology. In these physiologic roles, a de novo biosynthetic pathway has been ide ntified. However, the remodeling pathway is the one implicated in PAF inflammatory actions. Phospholipase A2 metabolizes phosphocholi ne precursors in cell membranes, releasing AA and PAF precursors, which are then acetylated into multiple species of PAE PAF release can be stimulated by various innate triggers, such as bacterial toxins, or trau ma and cytokines.

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