By Lionel Fry
Atopic eczema is the commonest and protracted dermatological noticeable regularly perform. during the last few years, advances were made within the genetics of atopy quite often, more recent remedies were brought for topical use, and new strategies instructed within the etiology. therefore, a brand new textual content on atopic eczema is either well timed and priceless. Written by way of one of many world's so much distinctive dermatologists, An Atlas of Atopic Eczema offers complete insurance of this dermis illness, together with new info on calcineurin inhibitors and their healing strategies. greatly illustrated in colour, the atlas explores issues resembling genetics, etiology, pathogenesis, diagnosis, medical beneficial properties, differential prognosis, relationships to different forms of eczema, and therapy. the writer lifts the veil of misunderstanding and is helping physicians remain on best of the problem the elevated occurrence of eczemas current.
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Extra resources for An Atlas of Atopic Eczema (Encyclopedia of Visual Medicine Series)
This is thought to be a more persistent form of the disease and carries a poorer prognosis. The lesions appear as red scaly areas (Figures 37 and 42), or crusting and weeping ones in the acute stages and thickened (lichenified) skin, particularly on the limbs, in the chronic phase (Figures 44 and 45). The lichenified patches are red or sometimes pale. The surface may be scaly and there is increased linearity of the skin surface markings. Excoriations (Figures 39, 44 and 45) are common and secondary infection, as seen in infants, may also occur (Figure 46).
This is usually due to Staphylococcus aureus but -hemolytic streptococcal infections may also occur. In infection with S. aureus, the lesions often develop golden crusts and/or weeping (Figure 35). It may be difficult to distinguish between acute weeping eczema (not infected) and secondarily infected eczema (Figure 36). Pustules are occasionally seen, making the diagnosis of secondary infection easier (Figure 36). In secondary infection with streptococci, erythema and edema of the skin surrounding the eczematous lesions are present.
If the lesions are asymmetrical, then this may imply secondary bacterial infection of the eczema. g. psoriasis, are as yet unknown. One possibility is that receptors in the endothelium of cutaneous vessels have developed symmetrically, as has the human body. In eczema and psoriasis, the rash is dependent on inflammatory cells coming into the skin via specialized receptors. Altered pigmentation Inflammation in the skin may either stimulate or suppress melanocyte function. Thus, the skin at the sites of the eczema may either become darker (Figures 59 and 60) or paler (Figures 61 and 62).
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